Birth Control With Least Mood Side Effects – If you are currently using hormonal contraceptives or have used them in the past, there is something you should know. These contraceptives cause more bone loss after menopause.

Women using these contraceptives experience bone loss at a time in their lives when they should be gaining bone mass!

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Bone mass typically increases from birth until about age 35, where it declines and then begins to decrease year by year for the rest of your life. So it’s very important to reach the highest possible bone mass before your mid-thirties. In addition to stopping the growth of bone mass, drugs that cause bone loss early in life can have devastating effects later in life.

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Including birth control pills, needles, implants, and some IUDs that cause bone loss. This is particularly worrying given that 67% of women using contraception use hormonal methods.

Hormonal contraceptives are devices or drugs that contain the hormones estrogen and progestin. In some cases, it may contain a combination of hormones, while in others it contains only progestin.

The primary use of hormonal contraceptives is fertility control, but they can also be taken to reduce ovarian cysts, acne, menstrual cramps, reduce flow, and regulate your menstrual cycle.

Hormonal contraceptives, which can cause amenorrhea, are now prescribed to girls under 14 years of age. Preventing normal menstrual cycles during adolescence results in bone loss at a time when major bone accretion should occur in a young woman’s life.

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The adverse effects of amenorrhea on skeletal mass and strength in the premenopausal woman may even result in severe premenopausal bone loss, especially if the amenorrhea begins in early adolescence and continues for several years. We fear losing bone mineral density

, (progestins are compounds created in the laboratory that produce progesterone-like effects), either alone or in combination with a synthetic estrogen-like compound. The two “estrogens” used with progestin in hormonal contraceptives are EE and mestranol, with EE being used more often.

Progestin drugs have varying degrees of estrogenic, androgenic, and anti-estrogenic effects and are thought to be responsible for some of the many adverse side effects caused by these drugs.

The first hormonal pill was approved in the 1960s. Since then, oral contraception has become popular around the world because of its relative ease of use.

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However, in addition to preventing the normal accumulation of bone mass in young women, hormonal contraceptives have shown the following side effects:

Why do hormonal contraceptives increase your risk for all these side effects? Hormonal contraceptives prevent the proper functioning of the endocrine glands

The endocrine system consists of endocrine glands that produce hormones. These glands are the messengers that keep your body working as it should! They are chemicals released into the bloodstream to direct processes such as sexual development, growth, and metabolism. They also help regulate your emotions.

The main glands of the female endocrine system are the hypothalamus, pituitary gland, pineal gland (in your brain), thyroid gland, parathyroid (in your neck), thymus (between your lungs), adrenal glands (above your kidneys, pancreas (behind your stomach)), and ovaries (in your pelvic area).

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If it is a progestin-only or progestin + “estrogen” contraceptive, it will disrupt the normal functioning of the HPA axis (located in the center of the brain and the hypothalamic-pituitary-ovarian axis). all endocrine functions). Interfering with healthy HPA axis directions can prevent ovulation.

Hormonal contraceptives kick the HPA axis into gear by suppressing the release of the two hormones that trigger ovulation and a woman’s production of estrogen and progesterone. When a woman takes hormonal contraceptives, these two hormones, luteinizing hormone [LH] and follicle-stimulating hormone [FSH], are not secreted, and the surge in LH production that should occur in the middle of the menstrual cycle does not occur. All this leads to ovulation, so ovulation does not occur and estrogen and progesterone are not produced.

Studies in women in many countries have shown that the use of all types of hormonal contraceptives is associated with significant losses in bone mineral density.

It remains unclear whether such losses increase the risk of fractures later in life. A young woman’s loss, rather than gain, of bone mineral density (BMD), which has been shown to occur in the spine, hip, and wrist, undoubtedly increases the risk of fractures in these regions, in addition to age-related bone loss. fields.

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A Cochrane Database Systematic Review was conducted with the hope of evaluating the completed research on fracture risk associated with the use of hormonal contraceptives. This review, which included studies on the effects of hormonal contraceptives on fracture risk and/or bone health, identified 19 randomized controlled trials published up to April 2014 that met the eligibility criteria. Eleven trials compared different combined oral contraceptives (COCs). These COCs contain an estrogen-like compound, EE, plus a progestin, such as depot medroxyprogesterone acetate [DMPA], norgestrel, levonorgestrel, Northindrone, etinodiol, etc. Click here for a complete list of progestins.

Another 8 trials examined other versions of these contraceptives: injectable forms, implant forms, IUD, transdermal patch and vaginal ring. Neither trial had fractures, instead they measured BMD and various biochemical markers of bone turnover. Seventeen of the studies measured BMD and 12 trials assessed biochemical markers of bone turnover.

Nevertheless, the reviewers said they could not draw strong conclusions from the mix of evidence and results. They noted that many of the trials had small numbers of participants; however, some women in these studies noted very large BMD losses.

Researchers are still working to see if postmenopausal women have an increased risk of severe bone weakening. A direct link between contraceptives and acutely low bone density later in life would require an intervention study in which two large groups of women (one on the pill and one on placebo) were followed for decades. Don’t hold your breath because this test will never happen! They will never have their research approved by institutional review boards because it is unethical to give women a placebo birth control pill to begin with.

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Because many women use oral contraceptives—many starting in early adolescence—many studies have examined the physiological changes these drugs produce. These include changes in general health (such as fluid retention, irritability, bloating, and weight gain), as well as changes in nutritional needs.

Numerous studies have investigated whether women on OCs are at risk of deficiencies in certain vitamins and minerals. The results seen in these studies led the World Health Organization (WHO) to publish a report that OCs contribute to the depletion of a number of essential nutrients, including the essential B vitamins (folate, vitamin B2 [riboflavin], B6 and B12), in addition to vitamins causes. Vitamin C, vitamin E and the minerals magnesium, selenium and zinc.

As far back as the 1960s, studies have shown that women who use OCs not only have lower levels of folic acid in their blood, but their folate levels are so low that they are deficient in this vital nutrient. In addition, blood folate levels continue to decrease as the duration of OC use increases. OCs cause folate deficiency by inhibiting folate absorption, increasing urinary excretion, and increasing the activity of folate-metabolizing enzymes.

Research from the 1970s showed that riboflavin deficiency was more common in women taking OC. Riboflavin deficiency leads to degenerative changes in the nervous system, endocrine dysfunction, skin disorders, and anemia—and may also be responsible for a frequently reported side effect of OCs—increased frequency, duration, and intensity of headaches.

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If you’re taking OCs and wondering why you’re often depressed, it may be because B6 deficiency causes a lack of serotonin, which promotes feelings of well-being (and whose production and/or maintenance is the target of many antidepressant drugs). The latest studies confirm that women taking OCs should take supplemental B6 to prevent deficiency.

OCs have been shown to prevent B12 from binding to transcobalamin, a transporter in the bloodstream, thus preventing B12 from being delivered throughout the body. Impaired B12 status in a pregnant woman greatly increases the risk of neural tube defects in her child.

Vitamin C levels in red blood cells (platelets) and immune cells (leukocytes) are reduced by the use of OCs, especially estrogen-like compounds. In addition, OCs increase

(the production of more free radicals than our cells can neutralize), so these drugs increase the need for vitamin C. Vitamin C is required for our collagen production. Since more than 90% of the protein in the bone matrix is ​​collagen, vitamin C is an essential nutrient for healthy bones.

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Combined OCs have been shown to reduce blood levels of vitamin E and increase platelet coagulation activity in healthy Caucasian women. Supplementation with vitamin E reversed this negative effect, leading researchers to hypothesize that the OC-related increase in heart attack and stroke risk may be due in part to the fact that these drugs reduce vitamin E levels. Natural vitamin E (mixed tocopherol and tocotrienol, NOT alpha-tocopherol alone) promotes healthy bones by neutralizing both bases.

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